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By Frank J. Dixon

Learn Institute of Scripps sanatorium, los angeles Jolla. study within the box. For investigators. 19 individuals, 6 U.S.

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Exp. Med. 180, 1591-1597. Rubin, D. , and Finley, D. The proteasome: A protein-degrading organelle? Curr. Biol. 8, 854-858. Rubin, D. , Young, R. , Goldberg, A. , and Finley, D. (1996). Identification of the gal4 suppressor Sugl as a subunit of the yeast 26s proteasome. Nature, 379, 655-657. , and Huibregtse, J. M. (1995). Protein ubiquitination involving an El-E2-E3 enzyme ubiquitin thioester cascade. Nature 373, 81-83. , and Baumeister, W. Proteasome from Themplasma acidqphilum: A threonine protease.

Rep. 21, 27-34. Eisenlohr, L. , Bennink, J. , and Yewdell, J. W. Expression of a membrane protease enhances presentation of endogenous antigens in MHC class I-restricted T lymphocytes. Cell 71, 963-972. , and Townsend, A. (1 995). Processing of major histocompatibility class I-restricted antigens in the endoplamic reticulum. J. Exp. Med. 181,14811491. Engehard, V. H. (1994). Structure of peptides associated with class I and class I1 MHC molecules. Annu. Rev. Immnunol. 12, 181-207. Fehling, H. J..

Med. 180, 1599-1607. , and Rechsteiner, M. (1995). Effects of interferon gamma and major histocompatibilitycomplex-encoded subunits on peptidase activities of human multicatalytic proteases. Proc. Natl. h a d . Sci. USA 92,584-588. , Ashton-Richardt, P. , Ploegh, H. , and Tonegawa, S. (1992). TAP1 mutant mice are deficient in antigen presentation, surface class I molecules, and CD4-8’ cells. Cell 71, 1205-1214. , Ashton-Richardt, P. , Rock, K. , Goldberg, A. , Doherty, P. , and Tonegawa, S. (1994).

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