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By John A. Smith

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Naito S, Walker SM, Fidler D (1989) In vivo selection of human renal cell carcinoma cells with high metastatic potential in nude mice. Clin Exptl Metastasis 7:381-389 12. Pliskin ME, Prehn RT (1975) Stimulation of liver regeneration and compensatory kidney hyperplasia by passive transfer of spleen cells. I Reticuloendothel Soc 17:290-299 13. Murphy GP, Tritsch GL, Sufrin G, Williams PD (1985) Kinetics of formation of renotropic growth factor. Urology 25:622-624 Oncogenes in Renal Cell Carcinoma E.

LB), with the common denominator being loss of chromosomal material from 3p [2]. Several molecular oncogene studies of RCC have led on from these cytogenetic observations. Deregulation of c-myc, which is located at 8q24, was suggested by the translocation breakpoint of 8q24 observed in familial RCC (Fig. lA); one hypothesis was that activation of c-myc by translocation, which is important in the genesis of Burkitt's lymphoma, might also be important in RCC. Although c-myc is frequently expressed in RCC [3-5], the level of expression appears to be no higher than in normal tissue [3, 6].

In Vitro 14:779-786 4. Vessella R, Moon T, Chiou R, Nowak J, Arfman E, Palme 0, Peterson G, Lange p (1985) Monoclonal annbodies to human renal cell carcinoma: Recognition of shared and restricted antigens. Cancer Res 45:6131-6139 5. Chiou RK, Vessella RL, Elson MK, Clayman RV, Gonzalez-Campoy J, Klicka MI, Shafer RB, Lange PH (1985) LocaIization of human renal cell carcinoma xenografts with a tumor-preferential monoclonal antibody. Cancer Res 45:6140-6146 6. Moon T, Vessella R, Palme 0, Nowak J, Lange P (1985) A highly restricted antigen for renal cell carcinoma defined by a monoclonal antibody.

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